Matches in Nanopublications for { ?s ?p "Jubilant: Expression of PTEN in both U87 MG and U373 MG resulted in the %reduction of BAD phosphorylation. expression of wild-type or substrate-trapping forms of PTEN in HEK293 cells altered the levels of the phospholipid products of phosphatidylinositol 3-kinase and ectopic expression of the phosphatase in PTEN-deficient tumor cell lines resulted in the inhibition of protein kinase (PK) B/Akt and regulation of cell survival." ?g. }
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- _5 value "Jubilant: Expression of PTEN in both U87 MG and U373 MG resulted in the %reduction of BAD phosphorylation. expression of wild-type or substrate-trapping forms of PTEN in HEK293 cells altered the levels of the phospholipid products of phosphatidylinositol 3-kinase and ectopic expression of the phosphatase in PTEN-deficient tumor cell lines resulted in the inhibition of protein kinase (PK) B/Akt and regulation of cell survival." provenance.
- _5 value "Jubilant: Expression of PTEN in both U87 MG and U373 MG resulted in the %reduction of BAD phosphorylation. expression of wild-type or substrate-trapping forms of PTEN in HEK293 cells altered the levels of the phospholipid products of phosphatidylinositol 3-kinase and ectopic expression of the phosphatase in PTEN-deficient tumor cell lines resulted in the inhibition of protein kinase (PK) B/Akt and regulation of cell survival." provenance.
- _5 value "Jubilant: Expression of PTEN in both U87 MG and U373 MG resulted in the %reduction of BAD phosphorylation. expression of wild-type or substrate-trapping forms of PTEN in HEK293 cells altered the levels of the phospholipid products of phosphatidylinositol 3-kinase and ectopic expression of the phosphatase in PTEN-deficient tumor cell lines resulted in the inhibition of protein kinase (PK) B/Akt and regulation of cell survival." provenance.
- _5 value "Jubilant: Expression of PTEN in both U87 MG and U373 MG resulted in the %reduction of BAD phosphorylation. expression of wild-type or substrate-trapping forms of PTEN in HEK293 cells altered the levels of the phospholipid products of phosphatidylinositol 3-kinase and ectopic expression of the phosphatase in PTEN-deficient tumor cell lines resulted in the inhibition of protein kinase (PK) B/Akt and regulation of cell survival." provenance.