Matches in Nanopublications for { ?s ?p "Multiple extracellular stimuli like peptide growth factors, steroid hormones, and environmental stresses activate AP-1 through JNK. c-Jun is a specific substrate and functional endpoint for JNK. Hence, by activating c-Jun, JNK actually controls various AP-1-related cellular processes such as proliferation, differentiation, transformation, apoptosis, and development. Proliferation of prostate cancer cell lines is highly correlated with serum-supported JNK activity, and growth of prostate carcinoma cells in vitro and in vivo requires JNK" ?g. }
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- _4 value "Multiple extracellular stimuli like peptide growth factors, steroid hormones, and environmental stresses activate AP-1 through JNK. c-Jun is a specific substrate and functional endpoint for JNK. Hence, by activating c-Jun, JNK actually controls various AP-1-related cellular processes such as proliferation, differentiation, transformation, apoptosis, and development. Proliferation of prostate cancer cell lines is highly correlated with serum-supported JNK activity, and growth of prostate carcinoma cells in vitro and in vivo requires JNK" provenance.
- _4 value "Multiple extracellular stimuli like peptide growth factors, steroid hormones, and environmental stresses activate AP-1 through JNK. c-Jun is a specific substrate and functional endpoint for JNK. Hence, by activating c-Jun, JNK actually controls various AP-1-related cellular processes such as proliferation, differentiation, transformation, apoptosis, and development. Proliferation of prostate cancer cell lines is highly correlated with serum-supported JNK activity, and growth of prostate carcinoma cells in vitro and in vivo requires JNK" provenance.