Matches in Nanopublications for { ?s ?p "Our results show that, although the deletion construct lacking NF-B (MMP9-608) was less responsive (p 0.05), the construct lacking both NF-B and AP-1 (MMP9-66) had activity close to that of the promoterless pGL3-Basic vector (p 0.001) (Fig. 5C), suggesting that both NF-B and AP-1 play a role in IL-18-mediated MMP9 transcription....These data demonstrate that IL-18 induces MMP9 transcription in SMCs via NF-B and AP-1 transcription factors (Fig. 5)." ?g. }
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- _4 value "Our results show that, although the deletion construct lacking NF-B (MMP9-608) was less responsive (p 0.05), the construct lacking both NF-B and AP-1 (MMP9-66) had activity close to that of the promoterless pGL3-Basic vector (p 0.001) (Fig. 5C), suggesting that both NF-B and AP-1 play a role in IL-18-mediated MMP9 transcription....These data demonstrate that IL-18 induces MMP9 transcription in SMCs via NF-B and AP-1 transcription factors (Fig. 5)." provenance.
- _4 value "Our results show that, although the deletion construct lacking NF-B (MMP9-608) was less responsive (p 0.05), the construct lacking both NF-B and AP-1 (MMP9-66) had activity close to that of the promoterless pGL3-Basic vector (p 0.001) (Fig. 5C), suggesting that both NF-B and AP-1 play a role in IL-18-mediated MMP9 transcription....These data demonstrate that IL-18 induces MMP9 transcription in SMCs via NF-B and AP-1 transcription factors (Fig. 5)." provenance.