Matches in Nanopublications for { ?s ?p "TNF-a, IL-6, and NO as well as iNOS or COX-2 are major mediators involved in systemic inflammation, and these mediators are induced readily in response to LPS (1)...The downregulation of 67LR did not affect LPSinduced production or expression of these mediators (Fig. 2D?H)." ?g. }
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- _3 value "TNF-a, IL-6, and NO as well as iNOS or COX-2 are major mediators involved in systemic inflammation, and these mediators are induced readily in response to LPS (1)...The downregulation of 67LR did not affect LPSinduced production or expression of these mediators (Fig. 2D?H)." provenance.
- _2 value "TNF-a, IL-6, and NO as well as iNOS or COX-2 are major mediators involved in systemic inflammation, and these mediators are induced readily in response to LPS (1)...The downregulation of 67LR did not affect LPSinduced production or expression of these mediators (Fig. 2D?H)." provenance.
- _3 value "TNF-a, IL-6, and NO as well as iNOS or COX-2 are major mediators involved in systemic inflammation, and these mediators are induced readily in response to LPS (1)...The downregulation of 67LR did not affect LPSinduced production or expression of these mediators (Fig. 2D?H)." provenance.
- _3 value "TNF-a, IL-6, and NO as well as iNOS or COX-2 are major mediators involved in systemic inflammation, and these mediators are induced readily in response to LPS (1)...The downregulation of 67LR did not affect LPSinduced production or expression of these mediators (Fig. 2D?H)." provenance.