Matches in Nanopublications for { ?s ?p "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." ?g. }
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- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.
- _3 value "When the cells were treated in the presence of hexamethonium the percentage of cells in S-phase dropped to 28% � 1.7% (in A549 cells) and 34% � 2.8% (in H23 cells). Further, nicotine-induced proliferation of A549, H23, H441, and H226 cells was significantly ablated by 2 ?7 subunit antagonists, ?-bungarotoxin and methylallyl aconitine (MAA).... Similar results were obtained in NHBEs and SAECs from normal lungs as well as HMEC-Ls (Figure 1B)." provenance.