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- source_evidence_literature type ECO_0000212 NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_assertion description "[Protein kinase C (PKC)-? and PKC-?, which are critical intermediary signaling molecules for CNI-induced tumorigenic pathway, formed complex with PRAS40; and we found that the CNI treatment increased the complex formation between PRAS40 and PKC, particularly (PKC)-?. Inhibition of PKC activity using pharmacological inhibitor markedly decreased H-Ras-induced phosphorylation of PRAS40.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_assertion evidence source_evidence_literature NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_assertion SIO_000772 21886838 NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_assertion wasDerivedFrom befree-20140225 NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_assertion wasGeneratedBy ECO_0000203 NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.
- befree-20140225 importedOn "2014-02-25" NP923176.RA-JmxmYz5Yn2MxYH8FOLTgQ5duzXzXoiMIJQ6uu9dR6k130_provenance.