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- source_evidence_literature type ECO_0000212 NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_assertion description "[At present, secondary resistance mechanisms associated with progression are better known: clonal selection of EGFR resistance mutation (T790M mutation in exon 20), amplification of transmembrane receptors for other growth factors (c-met, HER family, IGF1R, or AXL), downstream molecular alterations in EGFR signaling pathway (PI3K or PTEN), and epithelial-mesenchymal transition or transdifferentiation to small-cell cancer.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_assertion evidence source_evidence_literature NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_assertion SIO_000772 23911281 NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_assertion wasDerivedFrom befree-2016 NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_assertion wasGeneratedBy ECO_0000203 NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.
- befree-2016 importedOn "2016-02-19" NP1099066.RAF30zsmytf1feV7dcI9rXVePfjtKXRo_e1gEUfp4mjWg130_provenance.