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- source_evidence_literature type ECO_0000212 NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_assertion description "[GWASs also revealed that polymorphic variants of genes encoding proteinase 3 (PR3), the predominant antigenic target of ANCA in GPA, and its main inhibitor, alpha-1 antitrypsin, are highly associated with GPA and, even more significantly, with PR3-ANCA positivity (regardless of the clinical diagnosis); this emphasizes the central pathogenic role of PR3 and humoral autoimmunity in PR3-ANCA positive vasculitis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_assertion evidence source_evidence_literature NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_assertion SIO_000772 25523449 NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_assertion wasDerivedFrom befree-2016 NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_assertion wasGeneratedBy ECO_0000203 NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.
- befree-2016 importedOn "2016-02-19" NP1249383.RAcuBw-mm0fzUfZu7enc0sgAeSizjKi5a3C1FgRYa2Ypw130_provenance.