Matches in Nanopublications for { ?s ?p ?o <http://rdf.disgenet.org/resource/nanopub/NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance>. }
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- source_evidence_literature type ECO_0000212 NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_assertion description "[Proven or potential transforming factors include the allelic loss of the MEN-1 suppressor gene at 11q13, the still unknown factor(s) associated with atrophic corporal gastritis in which overexpression of BCL-2 likely plays a favouring role by prolonging ECL exposure to mitogens, and agents with still unclarified role, such as basic fibroblast growth factor, human chorionic gonadotropin-alpha and transforming growth factor-alpha.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_assertion evidence source_evidence_literature NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_assertion SIO_000772 9474860 NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_assertion wasDerivedFrom befree-2016 NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_assertion wasGeneratedBy ECO_0000203 NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.
- befree-2016 importedOn "2016-02-19" NP1389209.RAf6jW4dHQQ49nME-rgYl4QKJi5ud0-IEnriMVgMHjdIQ130_provenance.