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- source_evidence_literature type ECO_0000212 NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_assertion description "[However, when the knockout mice become obese with age, they develop fasting hyperglycemia and glucose intolerance, while neither fasting hyperglycemia nor glucose intolerance is evident in the aged knockout mice without obesity, suggesting that both the genetic defect in glucose-induced insulin secretion and the acquired insulin resistance due to environmental factors are necessary to develop diabetes in Kir6.2 knockout mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_assertion evidence source_evidence_literature NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_assertion SIO_000772 10868950 NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_assertion wasDerivedFrom befree-2016 NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_assertion wasGeneratedBy ECO_0000203 NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.
- befree-2016 importedOn "2016-02-19" NP287382.RAZllrKnJqVeZ0c3KipJol1mcnSUF74LY6I820UxdNQTE130_provenance.