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- source_evidence_literature type ECO_0000212 NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion description "[These data are supported by our previous findings that GC receptor levels critically influence GC sensitivity and imply that p16(INK4A) inactivation, in addition to allowing unrestricted proliferation, represents a mechanism by which lymphoid tumor cells might escape cell death triggered by endogenous GC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion evidence source_evidence_literature NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion SIO_000772 11441822 NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion wasDerivedFrom befree-20150227 NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion wasGeneratedBy ECO_0000203 NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- befree-20150227 importedOn "2015-02-27" NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.