Matches in Nanopublications for { ?s ?p ?o <http://rdf.disgenet.org/resource/nanopub/NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance>. }
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- source_evidence_literature type ECO_0000212 NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_assertion description "[Finally, dysregulation of p21WAF1/CIP1/MDA6 did not modify SB-mediated down-regulation of E2F-1 or c-Myc, but was associated with enhanced down-regulation of cyclins D1 and E. Together, these findings indicate that in U937 leukemia cells, p21WAF1/CIP1/MDA6 plays a critical functional role in SB-mediated G1 arrest and maturation, and suggest that cells displaying dysregulation of this CDKI respond to SB by engaging a default apoptotic program.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_assertion evidence source_evidence_literature NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_assertion SIO_000772 11408941 NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_assertion wasDerivedFrom befree-2016 NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_assertion wasGeneratedBy ECO_0000203 NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.
- befree-2016 importedOn "2016-02-19" NP321063.RAlqiM1cPU3p4SeJ6Kq2e47ThXt1831vbxS0MU6iGWhro130_provenance.