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- source_evidence_literature type ECO_0000212 NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_assertion description "[We investigated the possible involvement of several molecules, including c-ABL, platelet-derived growth factor receptor (PDGFR), cellular FADD-like interleukin-1 alpha-converting enzyme-like inhibitory protein (c-FLIP)(L/S), Fas-associated DD kinase, p53, p21(WAF1), proteins of B-cell leukemia/lymphoma 2 (Bcl-2) family and cytochrome c. Imatinib did not modulate the expression or activation of its own targets, such as c-ABL, PDGFRalpha and PDGFRbeta, but it did affect the expression of c-FLIP(L), BCL2-associated X protein (Bax) and Bcl-2.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_assertion evidence source_evidence_literature NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_assertion SIO_000772 16983347 NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_assertion wasDerivedFrom befree-2016 NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_assertion wasGeneratedBy ECO_0000203 NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.
- befree-2016 importedOn "2016-02-19" NP569988.RA1ncIcMwdp04MXBD9TRHtepZ0fPS3HaGppBV08mM9-4c130_provenance.