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- source_evidence_literature type ECO_0000212 NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- source_evidence_literature label "DisGeNET evidence - LITERATURE" NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- source_evidence_literature comment "Gene-disease associations inferred from text-mining the literature." NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion evidence source_evidence_literature NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion SIO_000772 18813785 NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion wasDerivedFrom befree-2016 NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion wasGeneratedBy ECO_0000203 NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.
- befree-2016 importedOn "2016-02-19" NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.