Matches in Nanopublications for { ?s ?p "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
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- NP510878.RAVWywQSWf_c_pWRBAdCXDfrwfuEBxqsNHM9S0cWyD2ro130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP510878.RAVWywQSWf_c_pWRBAdCXDfrwfuEBxqsNHM9S0cWyD2ro130_provenance.
- NP793262.RABssF4yuugyF8hjKXsASrcFg17HWQX9ZvFmcbJbsaX3I130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP793262.RABssF4yuugyF8hjKXsASrcFg17HWQX9ZvFmcbJbsaX3I130_provenance.
- assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP442512.RAv1t2pRJJa5umMv6yyV9K6y9xOv6bAPMz8XZFk8JI-iA130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP442512.RAv1t2pRJJa5umMv6yyV9K6y9xOv6bAPMz8XZFk8JI-iA130_provenance.
- assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP375044.RAe6lvlAO68d07eQF9jnhIlEK9bq6KYA2MvQ2yrJ-CqUM130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP375044.RAe6lvlAO68d07eQF9jnhIlEK9bq6KYA2MvQ2yrJ-CqUM130_provenance.
- NP375040.RAA6JVoi1NYaq-e6aJDaQK_LJMHwS69x6kPkEQCI0UcK8130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP375040.RAA6JVoi1NYaq-e6aJDaQK_LJMHwS69x6kPkEQCI0UcK8130_provenance.
- NP374992.RAK7jaijmnGjtriTE8wd69dxi1uzRc__GOvgbPQ_IHT3g130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP374992.RAK7jaijmnGjtriTE8wd69dxi1uzRc__GOvgbPQ_IHT3g130_provenance.
- NP694672.RA1I3S1ZlNwXJpln5nNxOBdnr11LxR7O1kce3kdDfIvgk130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP694672.RA1I3S1ZlNwXJpln5nNxOBdnr11LxR7O1kce3kdDfIvgk130_provenance.
- NP694673.RAxsJmNofWyFZh8lapUK7ZXWiXvvtXzVIpuNp4VL1Ra3w130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP694673.RAxsJmNofWyFZh8lapUK7ZXWiXvvtXzVIpuNp4VL1Ra3w130_provenance.
- NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_assertion description "[Collectively, these data suggest that activation of an HIF-1alpha-regulated glycolysis module is closely related to the aggressive phenotype of HCC, and that ENO1, a glycolysis module gene, might serve as a new target to circumvent HCC metastasis.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP694674.RA3FxM-uKGQTlFlH98TDq4EkkO57ffLmR-ULaAnAVB8aA130_provenance.