Matches in Nanopublications for { ?s ?p "[These data are supported by our previous findings that GC receptor levels critically influence GC sensitivity and imply that p16(INK4A) inactivation, in addition to allowing unrestricted proliferation, represents a mechanism by which lymphoid tumor cells might escape cell death triggered by endogenous GC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }
Showing items 1 to 3 of
3
with 100 items per page.
- assertion description "[These data are supported by our previous findings that GC receptor levels critically influence GC sensitivity and imply that p16(INK4A) inactivation, in addition to allowing unrestricted proliferation, represents a mechanism by which lymphoid tumor cells might escape cell death triggered by endogenous GC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." provenance.
- NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_assertion description "[These data are supported by our previous findings that GC receptor levels critically influence GC sensitivity and imply that p16(INK4A) inactivation, in addition to allowing unrestricted proliferation, represents a mechanism by which lymphoid tumor cells might escape cell death triggered by endogenous GC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP306232.RAd6yEk9PTXxvoxS4RSbjUQY3msGz--j4gPq4Nxi9V4Fw130_provenance.
- NP323040.RAzcOIU92qAv_8YXT6TkEjILN6ZeA3cd10qTXvsvnqsvo130_assertion description "[These data are supported by our previous findings that GC receptor levels critically influence GC sensitivity and imply that p16(INK4A) inactivation, in addition to allowing unrestricted proliferation, represents a mechanism by which lymphoid tumor cells might escape cell death triggered by endogenous GC.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP323040.RAzcOIU92qAv_8YXT6TkEjILN6ZeA3cd10qTXvsvnqsvo130_provenance.