Nanopublications LDF server

Matches in Nanopublications for { ?s ?p "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine."@en ?g. }

• NP1217037.RAeSQLPTvsnfVyCKCqS7gw609gcwzVhG4iDTsr1MHqxSo130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217037.RAeSQLPTvsnfVyCKCqS7gw609gcwzVhG4iDTsr1MHqxSo130_provenance.
• NP1217038.RAan4hIAy4avjUaSaVX5SpLFpT_hDGPoSaMdpIpIzIjpo130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217038.RAan4hIAy4avjUaSaVX5SpLFpT_hDGPoSaMdpIpIzIjpo130_provenance.
• NP1217039.RAWQOQEOF5_EVJNZro-FFbEz2P4V9B_qoMaEufDRdx0dQ130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217039.RAWQOQEOF5_EVJNZro-FFbEz2P4V9B_qoMaEufDRdx0dQ130_provenance.
• NP1217059.RAZ0u-CSNCi4t97k528yEprC6qE9EiwMZaab98VhFeuo4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217059.RAZ0u-CSNCi4t97k528yEprC6qE9EiwMZaab98VhFeuo4130_provenance.
• NP1217045.RAC2FzjXIv0Z6cKEl8QYwuBhpo3kXJ8M_0e0IfWYkyULs130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217045.RAC2FzjXIv0Z6cKEl8QYwuBhpo3kXJ8M_0e0IfWYkyULs130_provenance.
• NP1217048.RACRGTLEDWoWpI5QrfEnIHouIyiQ5kVxLNIwKcDpsqGe8130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217048.RACRGTLEDWoWpI5QrfEnIHouIyiQ5kVxLNIwKcDpsqGe8130_provenance.
• NP1217042.RApPpZnuEmeHHCAwMKrOcFPPO8fz-KkyGEtUBBm1tM0SI130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217042.RApPpZnuEmeHHCAwMKrOcFPPO8fz-KkyGEtUBBm1tM0SI130_provenance.
• NP1217044.RAnDFJr0fMMSYJZ1Gt33MR-inWnizke0Z1MierkaMIEAE130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217044.RAnDFJr0fMMSYJZ1Gt33MR-inWnizke0Z1MierkaMIEAE130_provenance.
• NP1217049.RApCFE3EF58DDhWbNg8ovvSjs12t5hoQIvLyjuNMn6cbw130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217049.RApCFE3EF58DDhWbNg8ovvSjs12t5hoQIvLyjuNMn6cbw130_provenance.
• NP1217051.RArvsJnlvZNzDKZj8PEa32dULCCCfPKUBowLzSAt1K3OM130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217051.RArvsJnlvZNzDKZj8PEa32dULCCCfPKUBowLzSAt1K3OM130_provenance.
• NP1217061.RAoLkXLSXTKAifxuzD4Dajt_xr1bf0LV3RNr_pl0cpiMk130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217061.RAoLkXLSXTKAifxuzD4Dajt_xr1bf0LV3RNr_pl0cpiMk130_provenance.
• NP1217041.RAOv8XVZdfca0DpQFxx3SP3RFpMpmZN0fkAG33xHYU9-A130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217041.RAOv8XVZdfca0DpQFxx3SP3RFpMpmZN0fkAG33xHYU9-A130_provenance.
• NP1217043.RAK-H8gBiKUKda_SwXfBHWSdlgLqD4skBAQvyL6Y0XZU4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217043.RAK-H8gBiKUKda_SwXfBHWSdlgLqD4skBAQvyL6Y0XZU4130_provenance.
• NP1217046.RAH47-KkmfnU1mIDHa_AiZW9RpBIWiio3etMriVowR2G4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217046.RAH47-KkmfnU1mIDHa_AiZW9RpBIWiio3etMriVowR2G4130_provenance.
• NP1217053.RAE_szqGVW8Cz2DXSb8oHz6WPCtOosdAbtjgdf6J1ypyg130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217053.RAE_szqGVW8Cz2DXSb8oHz6WPCtOosdAbtjgdf6J1ypyg130_provenance.
• NP1217054.RAMA0J-DJg1ava51sEpVCUOiGCASWgNYx3yqM_omGH4R4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217054.RAMA0J-DJg1ava51sEpVCUOiGCASWgNYx3yqM_omGH4R4130_provenance.
• NP1217055.RAKHmmP96ebtAJ3q84JGK49uHjZdVYna_FbYTs4v3utK0130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217055.RAKHmmP96ebtAJ3q84JGK49uHjZdVYna_FbYTs4v3utK0130_provenance.
• NP1217056.RAEtqvVSSWIMrFwHpWWkrCft5goN0A58e-mjXoAtL0MDU130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217056.RAEtqvVSSWIMrFwHpWWkrCft5goN0A58e-mjXoAtL0MDU130_provenance.
• NP1217035.RA4WQpmqK1UKDjgp-vP6titghPJ502hvNOT2f6CAPwVtY130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217035.RA4WQpmqK1UKDjgp-vP6titghPJ502hvNOT2f6CAPwVtY130_provenance.
• NP1217036.RA2pdyglT60j9y2h_a6BF0V0wxZ4IyFeRTotRmou4Vof4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217036.RA2pdyglT60j9y2h_a6BF0V0wxZ4IyFeRTotRmou4Vof4130_provenance.
• NP1217047.RA-lUgxOZ6PsbZ_AAIpKf1f3Yi_2sLfq6N53ZAfsfuym4130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217047.RA-lUgxOZ6PsbZ_AAIpKf1f3Yi_2sLfq6N53ZAfsfuym4130_provenance.
• NP1217050.RA7gS4f2n-SBqQEICN4097Q8yy0S_cwLKDw1CXw8xgbGg130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217050.RA7gS4f2n-SBqQEICN4097Q8yy0S_cwLKDw1CXw8xgbGg130_provenance.
• NP1217052.RA875mmBIh3u5Hmlt_9s2dzaZ2y17m_GyWE3rWxaRvnXo130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217052.RA875mmBIh3u5Hmlt_9s2dzaZ2y17m_GyWE3rWxaRvnXo130_provenance.
• NP1217057.RAzMxhgffCBX0TgCiLV-QZOCAWk3s-kLZT_2nSXR6Sukw130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217057.RAzMxhgffCBX0TgCiLV-QZOCAWk3s-kLZT_2nSXR6Sukw130_provenance.
• NP1217058.RA4V72VFG3Xgf-G_0d6SbuQvRfgScCju_ArvKC-fOSQH0130_assertion description "[Ubc13 was dispensable for transforming growth factor ? (TGF?)-induced SMAD activation but was required for activation of non-SMAD signaling via TGF?-activating kinase 1 (TAK1) and p38, whose activity controls expression of numerous metastasis promoting genes. p38 activation restored metastatic activity to Ubc13-deficient cells, and its pharmacological inhibition attenuated BCa metastasis in mice, suggesting it is a therapeutic option for metastatic BCa.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP1217058.RA4V72VFG3Xgf-G_0d6SbuQvRfgScCju_ArvKC-fOSQH0130_provenance.