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- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion type Assertion NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_head.
- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion description "[However, when the knockout mice become obese with age, they develop fasting hyperglycemia and glucose intolerance, while neither fasting hyperglycemia nor glucose intolerance is evident in the aged knockout mice without obesity, suggesting that both the genetic defect in glucose-induced insulin secretion and the acquired insulin resistance due to environmental factors are necessary to develop diabetes in Kir6.2 knockout mice.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine." NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_provenance.
- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion evidence source_evidence_literature NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_provenance.
- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion SIO_000772 10868950 NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_provenance.
- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion wasDerivedFrom befree-2016 NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_provenance.
- NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_assertion wasGeneratedBy ECO_0000203 NP287381.RAvFxzOKGa-uUpcf0aoKxqkcwG2awNDZr9KdjaahuT_5A130_provenance.