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{ <http://www.tkuhn.ch/bel2nanopub/RA2qrwjCpso098ZqxaItaYEG33yC0T8-8I9wOwqPieGQw#_4> ?p ?o ?g. }
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These results suggest that, apart from the effect of ThEA on amylase release, it also appears to be a vasoactive chemical mediator that triggers vasodilatation, modulating the course of inflammation
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