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{ <http://www.tkuhn.ch/bel2nanopub/RA7EPRdtEi4_H6268YoizSxzsQnepTU21YIW3IPFsmI4I#_6> ?p ?o ?g. }
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Cardiac hypertrophy induced by over-expression of IGF1R was completely blocked by a dominant negative PI3K(p110alpha) mutant, suggesting IGF1R promotes compensated cardiac hypertrophy in a PI3K(p110alpha) dependent manner.
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_6
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