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{ <http://www.tkuhn.ch/bel2nanopub/RA7NgXX9U7l14KW_figMsnB0p-_GMWcgYCfGQXkIETzEw#_5> ?p ?o ?g. }
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"
Sin1 over-expression enhanced osmotic stress-induced phosphorylation of ATF-2 and ATF-2-mediated transcription, whereas knockdown of Sin1 expression by siRNA suppressed these responses.
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