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{ <http://www.tkuhn.ch/bel2nanopub/RA8JHxOQTrdg0o-KjJTNycd3xrG56bvwwhb9PZsuIv3l8#_4> ?p ?o ?g. }
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B-ATF inhibits transcriptional activation of a reporter gene containing TRE sites in a dose-dependent manner, presumably by competing with Fos for Jun and forming transcriptionally inert Jun/B-ATF heterodimers.
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