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{ <http://www.tkuhn.ch/bel2nanopub/RA8Kk4KjA4MJTwLsehF-mZxb_twQQXY0Qs-k54G-mRXig#_4> ?p ?o ?g. }
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These results suggest that endotoxin-induced bacterial translocation, mucosal injury, and ileal permeability are mediated via activation of xanthine oxidase, and not through complement activation or the liberation of macrophage products.
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