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{ <http://www.tkuhn.ch/bel2nanopub/RA8sHysa0gjyjmpoiZNu6A-VaTAXbP9qgEivxZess6azs#_5> ?p ?o ?g. }
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Here we show that transcription from androgen-dependent target genes is inhibited by Hey1 and that expression of a constitutively active form of Notch is capable of repressing transactivation by the endogenous androgen receptor (AR).
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