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{ <http://www.tkuhn.ch/bel2nanopub/RA9464U-VxAKsWbl0f6-kfC79pK3NOtY6eBDMsnHFm4UA#_6> ?p ?o ?g. }
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"
Upon ligand treatment, progesterone receptor (PR) interacted preferentially with SRC-1, which recruited CBP and significantly enhanced acetylation at K5 of histone H4.
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