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- _5 value "from full text - In another study, ATG16L1 deficiency in mice macrophages produced high amounts of inflammatory cytokines IL-1beta and IL-18 when these macrophages were exposed to bacterial lipopolysaccharide [29]. The increased production of IL-1beta and IL-18 is due to Toll/IL-1 receptor domain-containing adaptor inducing IFN-beta (TRIF)-dependent activation of caspase-1; where caspase 1 is thought to cleave pro?IL-1beta to yield active cytokine [29]." provenance.
- _5 wasQuotedFrom 19903423 provenance.