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{ <http://www.tkuhn.ch/bel2nanopub/RAB8lFXZ0bEnrPMBjU1qbCbUJwhgIbr403Gp_SESgrgh4#_3> ?p ?o ?g. }
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Treatment of vascular smooth muscle cells with angiotensin II as well as overexpression of the constitutively active mutant RacL61 increased ROS release as assessed by
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_3
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