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{ <http://www.tkuhn.ch/bel2nanopub/RACGm9Sqrp9nuDVdRCrY8ZAIGgg729qey21b0A6K-N-mQ#_6> ?p ?o ?g. }
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Of potential functional relevance, the expression of Par-4 allows TNFalpha to induce apoptosis in NIH-3T3 cells. In addition, the down-regulation of Par-4 levels by oncogenic Ras sensitizes cells to TNFalpha-induced NF-kappaB activation.
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_6
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