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- _4 value "The inhibition of this oncogenic cooperation re-induces the expression of the death receptor and restores the sensitivity to FAS-L-mediated apoptosis [27,50]. In addition to its interaction with JUN, STAT3 cooperates with the noncanonical nuclear factor-kB (NF-kB) signaling pathway to regulate apoptosis [51]. STAT3 facilitates the processing of the p52 subunit from its p100 precursor and interacts with p52 on DNA. Consequently, the cooperation between these two transcription factors prevents cell death in response to genotoxic drugs." provenance.
- _4 wasQuotedFrom 17118707 provenance.