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{ <http://www.tkuhn.ch/bel2nanopub/RACaBZAyUh1QAYMTxccClLgFFyo0FQ-2lTCU919x9cWRM#_4> ?p ?o ?g. }
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In contrast, wild-type Egr2 or an Egr2 point mutant unable to bind the endogenous repressors Nab1/2 (caEgr2) supresses basal osteoclast apoptosis and rescues osteoclasts from apoptosis induced by MEK1/2 or Egr2 inhibition.
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