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{ <http://www.tkuhn.ch/bel2nanopub/RAEVv_rRGXETXc-wsmUIDrhur2ECV7X5Rn0ne3CWBuxRA#_2> ?p ?o ?g. }
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High steady-state levels of ROS then damage OXPHOS proteins, cardiolipin and mtDNA. This oxidative damage aggravates mitochondrial dysfunction to further augment electron leakage and ROS formation, thus leading to a vicious circle [2,6].
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