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{ <http://www.tkuhn.ch/bel2nanopub/RAGCwYA8nPtRrjdMr08ll3CCUcfbDc0OzEyYv8m3_z7RQ#_5> ?p ?o ?g. }
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Overexpression of a constitutively active mutant of ASK1 led to NF kappaB activation and cardiac hypertrophy. Activated ASK1-induced hypertrophy was abolished by inhibition of NF-kappaB activation
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