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{ <http://www.tkuhn.ch/bel2nanopub/RAKDjyvBc-sGv6CCQYC_qYwpQQK6-uZa5QIt9P4Dt3Hak#_3> ?p ?o ?g. }
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Overexpression of a constitutively active mutant of ASK1 led to NF kappaB activation and cardiac hypertrophy. Activated ASK1-induced hypertrophy was abolished by inhibition of NF-kappaB activation
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_3
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11815436
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