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{ <http://www.tkuhn.ch/bel2nanopub/RAKgiI4fb0oib5CmIrnEjE1h2SbyG3V6UdeN4lLClyMj4#_7> ?p ?o ?g. }
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Expression of wild-type PDK1 significantly inhibited H2O2-induced capase-3 activity and PARP cleavage in a dosedependent manner, whereas there was no effect in the presence of kinase-dead PDK1 (supplemental Fig. 4).
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