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{ <http://www.tkuhn.ch/bel2nanopub/RALfHkjKQfme5eApjZsz5Nvk7YYSsXzHlA5o_OiIw3jQw#_5> ?p ?o ?g. }
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Of potential functional relevance, the expression of Par-4 allows TNFalpha to induce apoptosis in NIH-3T3 cells. In addition, the down-regulation of Par-4 levels by oncogenic Ras sensitizes cells to TNFalpha-induced NF-kappaB activation.
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