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{ <http://www.tkuhn.ch/bel2nanopub/RAMCiPz94kbEaauc7TPjSynQN7f1co6LMQBEBSSHZTrL4#_6> ?p ?o ?g. }
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The association of MLK3 with POSH is substantially increased by LY294002 treatment (Fig. 5C). Thus, inhibition of endogenous Akt kinases enhances the ability of POSH to scaffold MLK3.
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