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{ <http://www.tkuhn.ch/bel2nanopub/RAMP3jGksx45XAlLsOpCc-hYCnGRZJer8GxsQ-WHJWK4w#_5> ?p ?o ?g. }
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AMPKalpha1 knockdown impaired agonist-mediated Akt phosphorylation, whereas Akt1 knockdown did not affect AMPK activation, thus suggesting that AMPK lies upstream of Akt in the pathway leading from receptor activation to eNOS stimulation.
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