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{ <http://www.tkuhn.ch/bel2nanopub/RANRR2f3ATR6OC0XwkRuWk3eg8uFvE0S5shbJR0b5YDCg#_5> ?p ?o ?g. }
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Enforced expression of constitutively active MKK1 or AKT vectors could restore Rad51 protein levels, which were inhibited by erlotinib, and decrease erlotinib-induced cytotoxicity
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