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{ <http://www.tkuhn.ch/bel2nanopub/RANufKClFRRCJdG3ANtKn23ucQ0wkVQ6jy_bCkDuKKjfY#_6> ?p ?o ?g. }
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Pretreatment of these cells with candesartan markedly suppressed the activation of ERKs (Fig. 2f). These results indicate that mechanical stress may activate ERKs in cardiomyocytes through the AT1 receptor even in the absence of AII.
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