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{ <http://www.tkuhn.ch/bel2nanopub/RAR5NgWJWASdsWwFwtxopt1519UEMdgekmCUrErhrEjB0#_7> ?p ?o ?g. }
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p53 appears to exert its inhibition of human AR by disrupting its amino- to carboxyl-terminal (N-to-C) interaction, which is thought to be responsible for the homodimerization of this receptor
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_7
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