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{ <http://www.tkuhn.ch/bel2nanopub/RARnnvcpK0v7Y0KS74z7e469f9t8r54UO45jUNxdsNzq0#_3> ?p ?o ?g. }
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Inactivation of CUL4A, L2DTL, PCNA, DDB1 or ROC1 induced p53 stabilization and growth arrest. L2DTL, PCNA and DDB1/CUL4A complexes were found to physically interact with p53 tumor suppressor and its regulator MDM2/HDM2
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