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{ <http://www.tkuhn.ch/bel2nanopub/RARtAkKAux-nuDvOMzm1MjX8k2xorAMR0UDkug_TVvBVg#_3> ?p ?o ?g. }
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Growth of HBECs was highly dependent on epidermal growth factor (EGF) and completely inhibited by EGF receptor (EGFR) tyrosine kinase inhibitors, which induced G1 arrest from full text - EGFR inhibitors used were gefitinib and erlotinib
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