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{ <http://www.tkuhn.ch/bel2nanopub/RAUzXlaGwxH2oemB4sM_yDMybntZY7cMkhGEFc637nJkk#_5> ?p ?o ?g. }
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Thus, in cardiomyocytes, as in skeletal muscle, FOXO3a activates an atrogene transcriptional program, which retards or prevents hypertrophy and is down-regulated by multiple physiological and pathological stimuli of myocyte growth.
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