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{ <http://www.tkuhn.ch/bel2nanopub/RAVbTzAow3hTcAJgUktMr-QC7lTZoG_fib7rmYUQAKnIU#_2> ?p ?o ?g. }
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In contrast, increased ROS production attributable to haploinsufficiency of SOD2 isoform reduced aconitase activity in both basal and agoniststimulated conditions and increased VSMC proliferation.88
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