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- _4 value "Prolactin (Prl), which also activates Jak2 after binding its cognate receptor, has been shown to cause an increase in ErbB2 phosphorylation. Moreover, blocking autocrine production of Prl in breast tumor cells led to a reduction in ErbB2's phosphotyrosine content and a decrease in MAPK activity [44]." provenance.
- _4 wasQuotedFrom 12648469 provenance.