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- _3 value "Meng et al. [23] conducted a study to determine whether co-exposure to smoke and LPS enhanced inflammatory response and other pathological effects related to COPD development in mice (Table 3). The most up-regulated genes in the three groups were Mmp12 (matrix metalloproteinase 12, 18.2-fold) in the smoke group, serum amyloid A1 in the LPS (242.4-fold), and LPS?smoke group (167.7- fold). Marco (macrophage receptor with collagenous structure) was also up-regulated by LPS (14.8-fold) and LPS?smoke (25.5-fold) groups. Noxo1 (NADPH oxidase organizer 1) was induced by all three treatment groups." provenance.
- _3 wasQuotedFrom 17888717 provenance.