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{ <http://www.tkuhn.ch/bel2nanopub/RA_2qdlaGzo6gBW_yjrHQa59czYLc15hdjsREyQ6z37uo#_5> ?p ?o ?g. }
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It has recently been shown that AML1, AML2, or both bind to their proposed binding sites in the Tbeta RE and form a complex with Smad3 to stimulate transcription from the GL Ig alpha promoter in a cooperative manner (13, 14).
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