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- _4 value "Tgfb1-mediated p38 activation relies on the kinase activity of Tgfbr1 but does not appear to involve a smad-mediated mechanism. This mutation activation could be blocked by Tgfbr1(KR), a kinase deficient mutant type I receptor. Tgfbr1 blocked both Tgfbr1 induced apoptosis and growth arrest." provenance.
- _4 wasQuotedFrom 12110587 provenance.