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{ <http://www.tkuhn.ch/bel2nanopub/RAa114lWgIjihQxCMQIQkj2PxvGRzST3YB16z7s9tANS0#_4> ?p ?o ?g. }
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(Stat1). The anti-inflammatory response of LPL in suppressing TNF-alpha-induced gene expression was a result of its inhibition of NF-kappaB activity by the abrogation of IkappaB-alpha degradation and phosphorylation of the p65 subunit.
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