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{ <http://www.tkuhn.ch/bel2nanopub/RAaFckDRIyF10dP00xtSjYLFgbl8dRkXX-aJDnPvsNZyA#_5> ?p ?o ?g. }
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However, under hypoxia AK1-deficient hearts, compared with the wild type, had a blunted AK-catalyzed phosphotransfer response, lowered intracellular ATP levels, increased P(i)/ATP ratio, and suppressed generation of adenosine
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